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anti-cav1.2a (cacna1c) antibody  (Alomone Labs)


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    Structured Review

    Alomone Labs anti-cav1.2a (cacna1c) antibody
    Anti Cav1.2a (Cacna1c) Antibody, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 13 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/cacna1c/Anti-CaV1%2E2a+(CACNA1C)+Antibody/custom%40acc-013%4042203078
    Average 94 stars, based on 13 article reviews
    anti-cav1.2a (cacna1c) antibody - by Bioz Stars, 2026-09
    94/100 stars

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    Related Articles

    Membrane:

    Article Title: Enhancement of airway ciliary beating mediated via voltage-gated Ca 2+ channels/α7-nicotinic receptors in mice.
    Article Snippet: Acetylcholine (ACh), which activates muscarinic ACh receptors (mAChRs) and nicotinic ACh receptors (nAChRs), enhances airway ciliary beating by increasing the intracellular Ca2+ concentration ([Ca]i).. The mechanisms enhancing airway ciliary beating by nAChRs have remained largely unknown, although those by mAChRs are well understood.. In this study, we focused on the effects of α7-nAChRs and voltage-gated Ca2+ channels (CaVs) on the airway ciliary beating.

    Incubation:

    Article Title: Variable Contribution of TMEM16A to Tone in Murine Arterial Vasculature.
    Article Snippet: .. Membranes were incubated overnight at 4°C in blocking buffer with primary antibody either against TMEM16A (1:500; Abcam #ab53212, UK), or CACNA1C (1:100; Alomone #ACC-003, Israel; pre-incubation with control antigen abolished bands at ~190 and ~210 kDa (not shown)) or pan-actin (1:1000; Cell Signalling Technology #4968, USA), or glyceraldehyde-3-phosphate dehydrogenase (GAPDH; 1:5000; Cell Signalling Technology #5174, A cc ep te d A rt ic le This article is protected by copyright. ..

    Article Title: Ion Channel Dysfunctions in Dilated Cardiomyopathy in Limb-Girdle Muscular Dystrophy
    Article Snippet: .. iPSC-CM were seeded in matrigel-coated 8-well Lab-Tek chambers (BD Biosciences) and incubated with the following primary antibodies at a dilution of 1:200: α-actinin (Sigma Aldrich), troponin T type 2 (Sigma Aldrich), CACNA1C (Alomone Labs), dystroglycan (Merk Millipore, VIA4-1 and IIH6C4), FKRP (Sigma Aldrich), and SCN5A (abcam). .. Nuclei were stained with Hoechst (Thermo fisher scientific).

    Article Title: Ion Channel Dysfunctions in Dilated Cardiomyopathy in Limb-Girdle Muscular Dystrophy
    Article Snippet: .. iPSC-CM were seeded in matrigel-coated eight-well Lab-Tek chambers (BD Biosciences) and incubated with the following primary antibodies at a dilution of 1:200: alpha-Actinin (Sigma Aldrich), TNNT (Sigma Aldrich), CACNA1C (Alomone Labs), Dystroglycan (Merk Millipore, VIA4-1 and IIH6C4), FKRP (Sigma Aldrich) and SCN5A (abcam). .. Nuclei were stained with Hoechst (Thermo fisher scientific).This was followed by incubation with Alexa Fluor 568-conjugated (Thermo Fisher Scientific) and Alexa Fluor 488-conjugated (Thermo Fisher Scientific) secondary antibodies.

    Blocking Assay:

    Article Title: Variable Contribution of TMEM16A to Tone in Murine Arterial Vasculature.
    Article Snippet: .. Membranes were incubated overnight at 4°C in blocking buffer with primary antibody either against TMEM16A (1:500; Abcam #ab53212, UK), or CACNA1C (1:100; Alomone #ACC-003, Israel; pre-incubation with control antigen abolished bands at ~190 and ~210 kDa (not shown)) or pan-actin (1:1000; Cell Signalling Technology #4968, USA), or glyceraldehyde-3-phosphate dehydrogenase (GAPDH; 1:5000; Cell Signalling Technology #5174, A cc ep te d A rt ic le This article is protected by copyright. ..

    Control:

    Article Title: Variable Contribution of TMEM16A to Tone in Murine Arterial Vasculature.
    Article Snippet: .. Membranes were incubated overnight at 4°C in blocking buffer with primary antibody either against TMEM16A (1:500; Abcam #ab53212, UK), or CACNA1C (1:100; Alomone #ACC-003, Israel; pre-incubation with control antigen abolished bands at ~190 and ~210 kDa (not shown)) or pan-actin (1:1000; Cell Signalling Technology #4968, USA), or glyceraldehyde-3-phosphate dehydrogenase (GAPDH; 1:5000; Cell Signalling Technology #5174, A cc ep te d A rt ic le This article is protected by copyright. ..

    Article Title: Embryo-derived trypsin-induced calcium entry is inhibited by endometrial infertility factor, LEFTY2
    Article Snippet: .. Membranes were probed overnight at 4°C with antibodies: LEFTY2 (1:500, sc-365845, Santa Cruz, Germany), CACNA1C (1:200, ACC-013, Alomone Labs, Israel), COX2 (1:200, MA5-14568, ThermoFisher, Germany), GAPDH (1:1,000, #2118L, Cell Signaling, Germany) was used for loading control. .. After 3 washes with TBS-T, each for 10 min, the membranes were incubated with HRP-conjugated anti-rabbit secondary antibody (1:2000, #7074s, Cell Signaling, Germany) or HRP-conjugated anti-mouse secondary antibody (1:2000, 7076S, Cell Signaling, Germany) at RT for 1 h, followed by 3 washes with TBS-T.

    Western Blot:

    Article Title: Loss of connectin novex-3 leads to heart dysfunction associated with impaired cardiomyocyte proliferation and abnormal nuclear mechanics.
    Article Snippet: .. :(0123456789) Scientific Reports | (2024) 14:13727 | https://doi.org/10.1038/s41598-024-64608-1 membranes were blocked with 5% nonfat milk in TBS/T, probed with primary antibodies for novex-3 (TTN2, Myomedix, designated as ab1 in Fig. 1a)16, connectin N-terminus (raised in our lab, designated as ab3 in Fig. 1a)44, Aurkb (A5102, Sigma-Aldrich), Nusap1 (12024-1-AP, Proteintech), Ccna2 (ZRB1590, Sigma-Aldrich), Atp2a2 (MA3-919, Thermo-Fisher), Cacna1c (ACC-003, Alomone), Slc8a1 (raised in our lab)45, Ryr2 (MA3-916, Thermo-Fisher), Pln (8495, Cell Signaling), and β-actin (sc81178, Santa Cruz), followed by secondary horseradish peroxidase (HRP)-conjugated IgG (Agilent Technologies, USA or GE Healthcare) and finally visualized by enhanced chemiluminescence (Western Lightning ECL-Pro; PerkinElmer, USA) using Amersham Imager 680 or LAS4000mini luminescent image analyser (GE Healthcare), as previously described46. ..

    Article Title: Atrial cardiomyocytes contribute to the inflammatory status associated with atrial fibrillation in right heart disease
    Article Snippet: CACNA1C, CX43, IL1β, IL6, KCNQ1, NLRP3, PLN, RYR2, SCN5A, TGFβ1, and GAPDH qPCR probes as well as DAPI and WGA were obtained from Invitrogen (Waltham, MA, USA). .. Primary antibodies for immunoblot experiments included: BIN1 (Novus Biologicals, NBP1-89102), CACNA1C (Alomone labs ACC-003), CAV (Novus Biologicals, NBP3-16503), IL6 (ThermoFisher ARC0962), JPH2 (ThermoFisher, PA5-141187), IL1β (ThermoFisher PA5-46956), KCNQ1 (NeuroMab clone N374/10), NLRP3 (Novus Biologicals NBP1-77080SS), SCN5A (Alomone labs ASC-005), RYR2 (Thermofisher MA3-916), SERCA2a (Thermofisher MA3-910). ..

    Article Title: Loss of connectin novex-3 leads to heart dysfunction associated with impaired cardiomyocyte proliferation and abnormal nuclear mechanics
    Article Snippet: The samples were then transferred onto nitrocellulose (Bio-Rad) or PVDF (GE Healthcare, USA) membranes using HorizeBLOT 2 M (WSE-4025, ATTO Corporation, Japan) at 20–25 V constant for 10–45 min, Trans-Blot Turbo (Bio-Rad) at 2.5 A constant for 3 min, or in the transfer buffer (25 mM Tris, 192 mM glycine and 20% methanol supplemented with or without 10 mM β-mercaptoethanol or 1% SDS) at 0.07–0.15 A constant for overnight. .. The membranes were blocked with 5% nonfat milk in TBS/T, probed with primary antibodies for novex-3 (TTN-2, Myomedix, designated as ab1 in Fig. a) , connectin N-terminus (raised in our lab, designated as ab3 in Fig. a) , Aurkb (A5102, Sigma-Aldrich), Nusap1 (12024-1-AP, Proteintech), Ccna2 (ZRB1590, Sigma-Aldrich), Atp2a2 (MA3-919, Thermo-Fisher), Cacna1c (ACC-003, Alomone), Slc8a1 (raised in our lab) , Ryr2 (MA3-916, Thermo-Fisher), Pln (8495, Cell Signaling), and β-actin (sc81178, Santa Cruz), followed by secondary horseradish peroxidase (HRP)-conjugated IgG (Agilent Technologies, USA or GE Healthcare) and finally visualized by enhanced chemiluminescence (Western Lightning ECL-Pro; PerkinElmer, USA) using Amersham Imager 680 or LAS4000mini luminescent image analyser (GE Healthcare), as previously described . ..



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    A Schematic illustrating imaging at the plasma membrane (PM). B Left: representative single-plane Airyscan confocal images of the PM showing Ca V 1.2 immunolabeling in control (CTL, black) and PFF-treated (red) neurons. Inset: MAP2 (pink) neuronal marker. Right: quantification of Ca V 1.2 cluster size, cluster density, and mean gray value (MGV) in the soma (a.) and dendrites (b.) of CTL (black) and PFF-treated (red) neurons. Dendritic measurements are shown separately for excitatory (dark blue) and inhibitory (light blue) populations. n = 20 somata per condition; n = 20 dendrites per group (CTL excitatory, CTL inhibitory, PFF excitatory, PFF inhibitory); two independent isolations with each isolation containing 8-10 pups. C Left: representative super-resolution TIRF localization maps showing Ca V 1.2 immunolabeling in CTL (black) and PFF-treated (red) neurons. Right: quantification of PM Ca V 1.2 cluster size, cluster density, and nearest-neighbor distance in the somatic region. n = 16 neurons per condition; two independent isolations. D Same experimental design as in ( B ), with neurons immunolabeled for Ca V 2.1. n = 19 (CTL) and n = 20 (PFF) somata; n = 20 dendrites per group (CTL excitatory, CTL inhibitory, PFF excitatory, PFF inhibitory); two independent isolations. E Same experimental design as in ( C ), with neurons immunolabeled for Ca V 2.1. n = 16 neurons per condition; two independent isolations. Error bars represent SEM. Statistical significance was determined using two-tailed Mann-Whitney or unpaired two-tailed t-tests. ns, not significant; *P ≤ 0.05; ***P ≤ 0.001; ****P ≤ 0.0001. CTL, control; PFF, α-synuclein pre-formed fibril treatment.
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    A Schematic illustrating imaging at the plasma membrane (PM). B Left: representative single-plane Airyscan confocal images of the PM showing Ca V 1.2 immunolabeling in control (CTL, black) and PFF-treated (red) neurons. Inset: MAP2 (pink) neuronal marker. Right: quantification of Ca V 1.2 cluster size, cluster density, and mean gray value (MGV) in the soma (a.) and dendrites (b.) of CTL (black) and PFF-treated (red) neurons. Dendritic measurements are shown separately for excitatory (dark blue) and inhibitory (light blue) populations. n = 20 somata per condition; n = 20 dendrites per group (CTL excitatory, CTL inhibitory, PFF excitatory, PFF inhibitory); two independent isolations with each isolation containing 8-10 pups. C Left: representative super-resolution TIRF localization maps showing Ca V 1.2 immunolabeling in CTL (black) and PFF-treated (red) neurons. Right: quantification of PM Ca V 1.2 cluster size, cluster density, and nearest-neighbor distance in the somatic region. n = 16 neurons per condition; two independent isolations. D Same experimental design as in ( B ), with neurons immunolabeled for Ca V 2.1. n = 19 (CTL) and n = 20 (PFF) somata; n = 20 dendrites per group (CTL excitatory, CTL inhibitory, PFF excitatory, PFF inhibitory); two independent isolations. E Same experimental design as in ( C ), with neurons immunolabeled for Ca V 2.1. n = 16 neurons per condition; two independent isolations. Error bars represent SEM. Statistical significance was determined using two-tailed Mann-Whitney or unpaired two-tailed t-tests. ns, not significant; *P ≤ 0.05; ***P ≤ 0.001; ****P ≤ 0.0001. CTL, control; PFF, α-synuclein pre-formed fibril treatment.
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    Image Search Results


    A Schematic illustrating imaging at the plasma membrane (PM). B Left: representative single-plane Airyscan confocal images of the PM showing Ca V 1.2 immunolabeling in control (CTL, black) and PFF-treated (red) neurons. Inset: MAP2 (pink) neuronal marker. Right: quantification of Ca V 1.2 cluster size, cluster density, and mean gray value (MGV) in the soma (a.) and dendrites (b.) of CTL (black) and PFF-treated (red) neurons. Dendritic measurements are shown separately for excitatory (dark blue) and inhibitory (light blue) populations. n = 20 somata per condition; n = 20 dendrites per group (CTL excitatory, CTL inhibitory, PFF excitatory, PFF inhibitory); two independent isolations with each isolation containing 8-10 pups. C Left: representative super-resolution TIRF localization maps showing Ca V 1.2 immunolabeling in CTL (black) and PFF-treated (red) neurons. Right: quantification of PM Ca V 1.2 cluster size, cluster density, and nearest-neighbor distance in the somatic region. n = 16 neurons per condition; two independent isolations. D Same experimental design as in ( B ), with neurons immunolabeled for Ca V 2.1. n = 19 (CTL) and n = 20 (PFF) somata; n = 20 dendrites per group (CTL excitatory, CTL inhibitory, PFF excitatory, PFF inhibitory); two independent isolations. E Same experimental design as in ( C ), with neurons immunolabeled for Ca V 2.1. n = 16 neurons per condition; two independent isolations. Error bars represent SEM. Statistical significance was determined using two-tailed Mann-Whitney or unpaired two-tailed t-tests. ns, not significant; *P ≤ 0.05; ***P ≤ 0.001; ****P ≤ 0.0001. CTL, control; PFF, α-synuclein pre-formed fibril treatment.

    Journal: bioRxiv

    Article Title: Nanoscale CaV channel reorganization links α-synuclein pathology to calcium-dependent transcriptional dysregulation

    doi: 10.64898/2026.05.01.719272

    Figure Lengend Snippet: A Schematic illustrating imaging at the plasma membrane (PM). B Left: representative single-plane Airyscan confocal images of the PM showing Ca V 1.2 immunolabeling in control (CTL, black) and PFF-treated (red) neurons. Inset: MAP2 (pink) neuronal marker. Right: quantification of Ca V 1.2 cluster size, cluster density, and mean gray value (MGV) in the soma (a.) and dendrites (b.) of CTL (black) and PFF-treated (red) neurons. Dendritic measurements are shown separately for excitatory (dark blue) and inhibitory (light blue) populations. n = 20 somata per condition; n = 20 dendrites per group (CTL excitatory, CTL inhibitory, PFF excitatory, PFF inhibitory); two independent isolations with each isolation containing 8-10 pups. C Left: representative super-resolution TIRF localization maps showing Ca V 1.2 immunolabeling in CTL (black) and PFF-treated (red) neurons. Right: quantification of PM Ca V 1.2 cluster size, cluster density, and nearest-neighbor distance in the somatic region. n = 16 neurons per condition; two independent isolations. D Same experimental design as in ( B ), with neurons immunolabeled for Ca V 2.1. n = 19 (CTL) and n = 20 (PFF) somata; n = 20 dendrites per group (CTL excitatory, CTL inhibitory, PFF excitatory, PFF inhibitory); two independent isolations. E Same experimental design as in ( C ), with neurons immunolabeled for Ca V 2.1. n = 16 neurons per condition; two independent isolations. Error bars represent SEM. Statistical significance was determined using two-tailed Mann-Whitney or unpaired two-tailed t-tests. ns, not significant; *P ≤ 0.05; ***P ≤ 0.001; ****P ≤ 0.0001. CTL, control; PFF, α-synuclein pre-formed fibril treatment.

    Article Snippet: For negative controls, Ca V 1.2 antibody (Alomone, ACC-003) was applied alone.

    Techniques: Imaging, Clinical Proteomics, Membrane, Immunolabeling, Control, Marker, Isolation, Two Tailed Test, MANN-WHITNEY

    A Schematic representation of the CCAD peptide mechanism of action. B Left: representative single-plane Airyscan confocal images of the PM in CTL and PFF-treated neurons co-incubated with SCRBL or CCAD peptides and co-immunolabeled for Ca V 1.2 and K V 2.1. Conditions are shown as CTL;SCRBL (black), PFF;SCRBL (red), CTL;CCAD (gray), and PFF;CCAD (yellow). Right: quantification of Ca V 1.2 cluster size, K V 2.1 cluster size, and Ca V 1.2-K V 2.1 overlap area in the somatic region. n = 19 (CTL;SCRBL), n = 19 (PFF;SCRBL), n = 20 (CTL;CCAD), and n = 20 (PFF;CCAD) neurons; two independent isolations. C Schematic representation of the proximity ligation assay (PLA). D Top: representative Airyscan confocal PLA images showing Ca V 1.2-K V 2.1 proximity. Images are maximum intensity projections from Z-stacks spanning whole cells. Color coding as in ( B ). Bottom: quantification of PLA puncta density. n = 18 neurons per condition; two independent isolations. E Same experimental design as in ( D ), but assessing Ca V 2.1-K V 2.1 proximity. n = 18 neurons per condition; two independent isolations. Error bars represent SEM. Statistical significance in panels ( B, D-E ) was determined using two-way ANOVA with appropriate post hoc tests. ns, not significant; **P ≤ 0.01; ***P ≤ 0.001; ****P ≤ 0.0001. CTL, control; PFF, α-synuclein pre-formed fibril treatment; CCAD, calcium channel association domain peptide; SCRBL, scrambled control peptide.

    Journal: bioRxiv

    Article Title: Nanoscale CaV channel reorganization links α-synuclein pathology to calcium-dependent transcriptional dysregulation

    doi: 10.64898/2026.05.01.719272

    Figure Lengend Snippet: A Schematic representation of the CCAD peptide mechanism of action. B Left: representative single-plane Airyscan confocal images of the PM in CTL and PFF-treated neurons co-incubated with SCRBL or CCAD peptides and co-immunolabeled for Ca V 1.2 and K V 2.1. Conditions are shown as CTL;SCRBL (black), PFF;SCRBL (red), CTL;CCAD (gray), and PFF;CCAD (yellow). Right: quantification of Ca V 1.2 cluster size, K V 2.1 cluster size, and Ca V 1.2-K V 2.1 overlap area in the somatic region. n = 19 (CTL;SCRBL), n = 19 (PFF;SCRBL), n = 20 (CTL;CCAD), and n = 20 (PFF;CCAD) neurons; two independent isolations. C Schematic representation of the proximity ligation assay (PLA). D Top: representative Airyscan confocal PLA images showing Ca V 1.2-K V 2.1 proximity. Images are maximum intensity projections from Z-stacks spanning whole cells. Color coding as in ( B ). Bottom: quantification of PLA puncta density. n = 18 neurons per condition; two independent isolations. E Same experimental design as in ( D ), but assessing Ca V 2.1-K V 2.1 proximity. n = 18 neurons per condition; two independent isolations. Error bars represent SEM. Statistical significance in panels ( B, D-E ) was determined using two-way ANOVA with appropriate post hoc tests. ns, not significant; **P ≤ 0.01; ***P ≤ 0.001; ****P ≤ 0.0001. CTL, control; PFF, α-synuclein pre-formed fibril treatment; CCAD, calcium channel association domain peptide; SCRBL, scrambled control peptide.

    Article Snippet: For negative controls, Ca V 1.2 antibody (Alomone, ACC-003) was applied alone.

    Techniques: Incubation, Immunolabeling, Proximity Ligation Assay, Control

    A Left: representative FV4000 confocal images showing co-immunolabeling of CDK5 and K V 2.1 at the PM in control (CTL, black) and PFF-treated (red) neurons. Images are maximum intensity projections from three optical sections acquired at the PM. Right: quantification of CDK5-K V 2.1 overlap area and CDK5 puncta density in the somatic region. n = 18 (CTL) and n = 19 (PFF) neurons; two independent isolations. B Schematic representation of the roscovitine mechanism of action. C Top: representative FV4000 confocal images of the PM in CTL and PFF-treated neurons incubated with or without roscovitine and immunolabeled for pS603-K V 2.1. Conditions are shown as CTL (black), PFF (red), CTL;Rosco (blue), and PFF;Rosco (purple). Images are maximum intensity projections from Z-stacks spanning whole cells. Bottom: quantification of somatic pS603-Kv2.1 occupancy (% of soma area), cluster MGV, and cluster density. n = 19 (CTL), n = 20 (PFF), n = 20 (CTL;Rosco), and n = 20 (PFF;Rosco) neurons; two independent isolations. D Left: representative FV4000 confocal PLA images showing Ca V 1.2-K V 2.1 proximity. Images are maximum intensity projections from Z-stacks spanning whole cells. Color coding as in ( C ). Right: quantification of PLA puncta density. n = 20 neurons per condition; two independent isolations. E Same experimental design as in ( D ), but assessing Ca V 2.1-K V 2.1 proximity. n = 20 (CTL), n = 20 (PFF), n = 21 (CTL;Rosco), and n = 20 (PFF;Rosco) neurons; two independent isolations. Error bars represent SEM. Statistical significance in panel ( A ) was determined using two-tailed Mann-Whitney test; panels ( C – E ) were analyzed using two-way ANOVA with appropriate post hoc tests. ns, not significant; **P ≤ 0.01; ***P ≤ 0.001; ****P ≤ 0.0001. CTL, control; PFF, α-synuclein pre-formed fibril treatment; Rosco, roscovitine; pS603-K V 2.1, K V 2.1 phosphorylated at serine 603.

    Journal: bioRxiv

    Article Title: Nanoscale CaV channel reorganization links α-synuclein pathology to calcium-dependent transcriptional dysregulation

    doi: 10.64898/2026.05.01.719272

    Figure Lengend Snippet: A Left: representative FV4000 confocal images showing co-immunolabeling of CDK5 and K V 2.1 at the PM in control (CTL, black) and PFF-treated (red) neurons. Images are maximum intensity projections from three optical sections acquired at the PM. Right: quantification of CDK5-K V 2.1 overlap area and CDK5 puncta density in the somatic region. n = 18 (CTL) and n = 19 (PFF) neurons; two independent isolations. B Schematic representation of the roscovitine mechanism of action. C Top: representative FV4000 confocal images of the PM in CTL and PFF-treated neurons incubated with or without roscovitine and immunolabeled for pS603-K V 2.1. Conditions are shown as CTL (black), PFF (red), CTL;Rosco (blue), and PFF;Rosco (purple). Images are maximum intensity projections from Z-stacks spanning whole cells. Bottom: quantification of somatic pS603-Kv2.1 occupancy (% of soma area), cluster MGV, and cluster density. n = 19 (CTL), n = 20 (PFF), n = 20 (CTL;Rosco), and n = 20 (PFF;Rosco) neurons; two independent isolations. D Left: representative FV4000 confocal PLA images showing Ca V 1.2-K V 2.1 proximity. Images are maximum intensity projections from Z-stacks spanning whole cells. Color coding as in ( C ). Right: quantification of PLA puncta density. n = 20 neurons per condition; two independent isolations. E Same experimental design as in ( D ), but assessing Ca V 2.1-K V 2.1 proximity. n = 20 (CTL), n = 20 (PFF), n = 21 (CTL;Rosco), and n = 20 (PFF;Rosco) neurons; two independent isolations. Error bars represent SEM. Statistical significance in panel ( A ) was determined using two-tailed Mann-Whitney test; panels ( C – E ) were analyzed using two-way ANOVA with appropriate post hoc tests. ns, not significant; **P ≤ 0.01; ***P ≤ 0.001; ****P ≤ 0.0001. CTL, control; PFF, α-synuclein pre-formed fibril treatment; Rosco, roscovitine; pS603-K V 2.1, K V 2.1 phosphorylated at serine 603.

    Article Snippet: For negative controls, Ca V 1.2 antibody (Alomone, ACC-003) was applied alone.

    Techniques: Immunolabeling, Control, Incubation, Two Tailed Test, MANN-WHITNEY